e.g. mhealth
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Skip search results from other journals and go to results- 21 Journal of Medical Internet Research
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This is the peer-review report for “Identifying Safeguards Disabled by Epstein-Barr Virus Infections in Genomes From Patients With Breast Cancer: Chromosomal Bioinformatics Analysis.”
JMIRx Med 2025;6:e70041
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This is the peer-review report for “Identifying Safeguards Disabled by Epstein-Barr Virus Infections in Genomes From Patients With Breast Cancer: Chromosomal Bioinformatics Analysis.”
Title: “Herpesvirus infections eliminate safeguards against breast cancer and its metastasis: comparable to hereditary breast cancers”
The paper [1] hypothesizes that Epstein-Barr virus (EBV) infections promote breast cancer by disabling cancer safeguards.
JMIRx Med 2025;6:e70039
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This is the author’s response to peer-review reports for “Identifying Safeguards Disabled by Epstein-Barr Virus Infections in Genomes From Patients With Breast Cancer: Chromosomal Bioinformatics Analysis.”
Title: “Herpesvirus infections eliminate safeguards against breast cancer and its metastasis: comparable to hereditary breast cancers”
The paper [2] hypothesizes that Epstein-Barr virus (EBV) infections promote breast cancer by disabling cancer safeguards.
JMIRx Med 2025;6:e69307
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A human cancer virus is one such risk factor. A tumor virus does not cause cancer by itself [3] but can make cancer more likely by inhibiting tumor suppressors [4] or activating oncogenes. Viral damage then increases cancer risks via mutations and chromosome breaks. Epstein-Barr virus (EBV), also called human herpesvirus 4, infects at least 90% of humans as a lifelong infection, often acquired at an early age [5], but the virus remains latent and asymptomatic in most people.
JMIRx Med 2025;6:e50712
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An example of this can be seen in satellite viruses (eg, hepatitis delta virus, satellite tobacco necrosis virus 1) that encode ribozymes that are folded by pseudoknotted structures [8,9].
For decades, scientists have explored and created different prediction software to better elucidate the complicated nature of RNA folding.
JMIRx Bio 2024;2:e58899
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Structural and functional analyses of the lineage characterizing mutations in the spike glycoprotein have predicted potential alterations in virus-host interactions and masking of the antibody binding sites, leading to increased transmissibility, lethality, and immune escape capabilities for this variant [2-4], which were further validated in recent animal models [5,6] and human studies [7-10].
Interact J Med Res 2024;13:e44492
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Assessing SARS-CoV-2 Testing Adherence in a University Town: Recurrent Event Modeling Analysis
Both the virus itself and protective behaviors changed throughout the waves of the pandemic [1], complicating long-term interventions. Government and public health officials implemented health contingencies that were adaptable and flexible to reduce socioeconomic and long-term health burden fatigue [15,16].
Understanding how peoples’ adherence to preventive measures changes over time can guide policy makers as they amend strategies to revitalize public health strategies in future outbreaks.
JMIR Public Health Surveill 2024;10:e48784
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Every winter, respiratory syncytial virus (RSV) causes acute lower respiratory tract infections in approximately 33.8 million children younger than 5 years worldwide [1]. Approximately all children are infected at least once, and half are infected twice or more by the age of 24 months [2]. Newborns and children with underlying medical conditions are particularly susceptible to severe infection [3-5].
JMIR Form Res 2024;8:e52412
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In total, 9 different respiratory viruses were detected: human metapneumovirus type B (HMPV-B; detected in F02, F07, F09, and F21); respiratory syncytial virus type A (RSV-A; detected in F15); respiratory syncytial virus type B (RSV-B; detected in F13); human rhinovirus (HRV) types A40 (detected in F06), A80 (detected in F04), C42 (detected in F03 and F21), C45 (detected in F02 and F18), and C54 (detected in F06); and a diverse HRV-C most similar to HRV-C36 (detected in F19).
JMIR Form Res 2023;7:e32848
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